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Regulation of BRCA1 by protein degradation

Author
BLAGOSKLONNY, M. V1 ; AN, W. G2 ; MELILLO, G1 ; NGUYEN, P1 ; TREPEL, J. B1 ; NECKERS, L. M2
[1] Department of Therapeutics, Medicine Branch, National Cancer Institute, NIH, Bethesda, Maryland, MD 20892, United States
[2] Department of Cell and Cancer Biology, Medicine Branch, National Cancer Institute, NIH, Bethesda, Maryland, MD 20892, United States
Source

Oncogene (Basingstoke). 1999, Vol 18, Num 47, pp 6460-6468 ; ref : 54 ref

ISSN
0950-9232
Scientific domain
Cell biology, histology; Medical oncology; Genetics
Publisher
Nature Publishing, Basingstoke
Publication country
United Kingdom
Document type
Article
Language
English
Keyword (fr)
Carcinogenèse Cathepsin Glande mammaire Gène suppresseur tumeur Homme Lignée cellulaire Multicatalytic endopeptidase complex Protéolyse Régulation Tumeur Gène BRCA1 Protéine BRCA1 Cysteine endopeptidases Enzyme Hydrolases Peptidases
Keyword (en)
Carcinogenesis Cathepsin Mammary gland Tumor suppressor gene Human Cell line Multicatalytic endopeptidase complex Proteolysis Regulation(control) Tumor Cysteine endopeptidases Enzyme Hydrolases Peptidases
Keyword (es)
Carcinogénesis Cathepsin Glándula mamaria Gen supresor tumor Hombre Línea celular Multicatalytic endopeptidase complex Proteolisis Regulación Tumor Cysteine endopeptidases Enzima Hydrolases Peptidases
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
1210609

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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