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Insulin activation of phosphatidylinositol 3-kinase in the hypothalamic arcuate nucleus: A key mediator of insulin-induced anorexia

Author
NISWENDER, Kevin D1 ; MORRISON, Christopher D1 ; CLEGG, Deborah J2 ; OLSON, Ryan3 ; BASKIN, Denis G1 4 ; MYERS, Martin G3 ; SEELEY, Randy J2 ; SCHWARTZ, Michael W1
[1] Division of Metabolism, Endocrinology and Nutrition, University of Washington School of Medicine and Harborview Medical Center, Seattle, Washington, United States
[2] Department of Psychiatry, University of Cincinnati, Cincinnati, Ohio, United States
[3] Joslin Diabetes Center, Harvard Medical School, Boston, Massachusetts, United States
[4] Puget Sound Veterans Affairs Medical Center, Seattle, Washington, United States
Source

Diabetes (New York, NY). 2003, Vol 52, Num 2, pp 227-231, 5 p ; ref : 25 ref

CODEN
DIAEAZ
ISSN
0012-1797
Scientific domain
Endocrinology; Nutrition, obesity, metabolic disorders
Publisher
American Diabetes Association, Alexandria, VA
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Anorexie Insuline Noyau arqué Transduction signal Encéphale Hormone pancréatique Hypothalamus Système nerveux central
Keyword (en)
Anorexia Insulin Arcuate nucleus Signal transduction Brain (vertebrata) Pancreatic hormone Hypothalamus Central nervous system
Keyword (es)
Anorexia Insulina Núcleo arqueado Transducción señal Encéfalo Hormona pancreática Hipotálamo Sistema nervioso central
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A28 Vertebrates: endocrinology / 002A28A Hypothalamus. Hypophysis. Epiphysis. Urophysis / 002A28A02 Hormones and neuropeptides. Regulation

Discipline
Vertebrates : endocrinology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
14523253

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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