Mdm2 ligase dead mutants did not act in a dominant negative manner to re-activate p53, but promoted tumor cell growth
- Author
- SWAROOP, Manju1 ; YI SUN1
[1] Cancer Molecular Sciences, Pfizer Global Research and Development, Ann Arbor Laboratories, 2800 Plymouth Road, Ann Arbor, MI 48105, United States - Source
Anticancer research. 2003, Vol 23, Num 4, pp 3167-3174, 8 p ; ref : 32 ref
- ISSN
- 0250-7005
- Scientific domain
- Medical oncology
- Publisher
- International Institute of Anticancer Research, Attiki
- Publication country
- Greece
- Document type
- Article
- Language
- English
- Author keyword
- Apoptosis E3 ubiquitin ligase Mdm2 dominant negative p53 transcription
- Keyword (fr)
- Adénocarcinome Glande mammaire Gène TP53 Gène onc cellulaire Gène suppresseur tumeur Homme Ligases Lignée cellulaire établie Multiplication cellulaire Mutation Protooncogène Ubiquitine Gène mdm2 Lignée MCF 7 Enzyme Glande mammaire pathologie Génétique Tumeur maligne
- Keyword (en)
- Adenocarcinoma Mammary gland TP53 Gene C-Onc gene Tumor suppressor gene Human Ligases Established cell line Cell proliferation Mutation Protooncogene Ubiquitin Enzyme Mammary gland diseases Genetics Malignant tumor
- Keyword (es)
- Adenocarcinoma Glándula mamaria Gen TP53 Gen onc celular Gen supresor tumor Hombre Ligases Línea celular establecida Multiplicación celular Mutación Protooncogen Ubiquitina Enzima Glándula mamaria patología Genética Tumor maligno
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B04 Tumors / 002B04G Tumor cell / 002B04G01 General aspects (metabolism, cell proliferation, established cell line...)
- Discipline
- Tumours
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 15005593
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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