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Fas preassociation required for apoptosis signaling and dominant inhibition by pathogenic mutations

Author
SIEGEL, R. M1 ; FREDERIKSEN, J. K1 ; ZACHARIAS, D. A2 ; CHAN, F. K.-M1 ; JOHNSON, M1 ; LYNCH, D3 ; TSIEN, R. Y2 ; LENARDO, M. J1
[1] Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892, United States
[2] Howard Hughes Medical Institute and Department of Pharmacology, University of California, San Diego School of Medicine, La Jolla, CA 92093, United States
[3] Immunex Corporation, 51 University Street, Seattle, WA 98101, United States
Source

Science (Washington, D.C.). 2000, Vol 288, Num 5475, pp 2354-2357

CODEN
SCIEAS
ISSN
0036-8075
Scientific domain
Multidisciplinary
Publisher
American Association for the Advancement of Science, Washington, DC
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Apoptose Homme Inhibition Lymphoprolifératif syndrome Mutation Oligomérisation Récepteur biologique Transduction signal Protéine Fas Hémopathie maligne
Keyword (en)
Apoptosis Human Inhibition Lymphoproliferative syndrome Mutation Oligomerization Biological receptor Signal transduction Malignant hemopathy
Keyword (es)
Apoptosis Hombre Inhibición Linfoproliferativo síndrome Mutación Oligomerización Receptor biológico Transducción señal Hemopatía maligna
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04F Cell structures and functions / 002A04F03 Cell receptors / 002A04F03F Miscellaneous

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
1512276

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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