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Transcriptional repression of atherogenic inflammation: Modulation by PPARδ

Author
LEE, Chih-Hao1 ; CHAWLA, Ajay1 ; URBIZTONDO, Ned1 ; LIAO, Debbie1 ; BOISVERT, William A2 ; EVANS, Ronald M1
[1] Howard Hughes Medical Institute, Gene Expression Laboratory, Salk Institute for Biological Studies, 10010 North Torrey Pines Road, La Jolla, CA 92037, United States
[2] Vascular Medicine Research, Brigham & Women's Hospital, 65 Landsdowne Street, Room 275, Cambridge, MA 02139, United States
Source

Science (Washington, D.C.). 2003, Vol 302, Num 5644, pp 453-457, 5 p

CODEN
SCIEAS
ISSN
0036-8075
Scientific domain
Multidisciplinary
Publisher
American Association for the Advancement of Science, Washington, DC
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Animal Athérosclérose Inflammation Macrophage Récepteur PPAR-β Régulation Répresseur transcription Souris Appareil circulatoire pathologie Mammalia Rodentia Vaisseau sanguin pathologie Vertebrata
Keyword (en)
Animal Atherosclerosis Inflammation Macrophage PPAR-β receptor Regulation(control) Transcription repressor Mouse Cardiovascular disease Mammalia Rodentia Vascular disease Vertebrata
Keyword (es)
Animal Ateroesclerosis Inflamación Macrófago Receptor PPAR-β Regulación Represor transcripción Ratón Aparato circulatorio patología Mammalia Rodentia Vaso sanguíneo patología Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B12 Cardiology. Vascular system / 002B12B Blood and lymphatic vessels / 002B12B01 Atherosclerosis (general aspects, experimental research)

Discipline
Cardiology. Circulatory system
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
15228123

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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