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Nrf2 regulates the sensitivity of death receptor signals by affecting intracellular glutathione levels

Author
MORITO, Naoki1 ; YOH, Keigyou2 ; ITOH, Ken1 3 ; HIRAYAMA, Aki2 ; KOYAMA, Akio2 ; YAMAMOTO, Masayuki1 3 ; TAKAHASHI, Satoru1 4
[1] Institute of Basic Medical Sciences, University of Tsukuba, 1-1-1 Tennodai, Tsukuba, Ibaraki 305-8575, Japan
[2] Institute of Clinical Medicine, University of Tsukuba, 1-1-1 Tennodai, Tsukuba, Ibaraki 305-8575, Japan
[3] Center for Tsukuba Advanced Research Alliance, University of Tsukuba, 1-1-1 Tennodai, Tsukuba, Ibaraki 305-8575, Japan
[4] Laboratory Animal Resource Center, University of Tsukuba, 1-1-1 Tennodai, Tsukuba, Ibaraki 305-8575, Japan
Source

Oncogene (Basingstoke). 2003, Vol 22, Num 58, pp 9275-9281, 7 p ; ref : 40 ref

ISSN
0950-9232
Scientific domain
Cell biology, histology; Medical oncology; Genetics
Publisher
Nature Publishing, Basingstoke
Publication country
United Kingdom
Document type
Article
Language
English
Keyword (fr)
Apoptose Facteur nécrose tumorale Intracellulaire Mort Récepteur biologique Sensibilité Stress oxydatif
Keyword (en)
Apoptosis Tumor necrosis factor Intracellular Death Biological receptor Sensitivity Oxidative stress
Keyword (es)
Apoptosis Factor necrosis tumoral Intracelular Muerte Receptor biológico Sensibilidad Estrés oxidativo
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H05 Ageing, cell death

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
15399008

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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