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Nrf2 deficiency improves autoimmune nephritis caused by the fas mutation lpr

Author
MORITO, Naoki; YOH, Keigyou; HIRAYAMA, Aki; ITOH, Ken; NOSE, Masato; KOYAMA, Akio; YAMAMOTO, Masayuki; TAKAHASHI, Satoru
Institute for Basic Medicine, Ehime University, Ehime, Japan
Institute of Basic Medical Sciences, Institute of Clinical Medicine and Center for Tsukuba Advanced Research Alliance, University of Tsukuba, Tsukuba Ibaraki, Japan
Conference title
Forefronts in nephrology: immunology in renal diseases
Conference name
Forefronts in Nephrology Symposium Immunology in Renal Disease (Berlin 2003-06-02)
Author (monograph)
GOLDMAN, Michel (Editor); SCHÖCKLMANN, Harald (Editor); KUNZENDORF, Ulrich (Editor)
International Society of Nephrology, International (Funder/Sponsor)
Source

Kidney international. 2004, Vol 65, Num 5, pp 1703-1713, 11 p ; ref : 50 ref

CODEN
KDYIA5
ISSN
0085-2538
Scientific domain
Urology, nephrology
Publisher
Nature Publishing, New York, NY
Publication country
United States
Document type
Conference Paper
Language
English
Author keyword
Fas Nrf2 TNF-u apoptosis glutathione oxidative stress
Keyword (fr)
Apoptose Déficit Glutathion Mutation Néphropathie Stress oxydatif Urologie Néphrologie Appareil urinaire pathologie Rein pathologie
Keyword (en)
Apoptosis Deficiency Glutathione Mutation Nephropathy Oxidative stress Urology Nephrology Urinary system disease Kidney disease
Keyword (es)
Apoptosis Déficiencia Glutatión Mutación Nefropatía Estrés oxidativo Urología Nefrología Aparato urinario patología Riñón patología
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B14 Nephrology. Urinary tract diseases

Discipline
Nephrology. Urinary tract diseases
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
15710276

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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