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Inhibition of glucose-stimulated activation of extracellular signal-regulated protein kinases 1 and 2 by epinephrine in pancreatic β-cells

Author
GIBSON, Tara Beers1 ; LAWRENCE, Michael C1 ; EHRLICH, Barbara E2 ; COBB, Melanie H1 ; GIBSON, Craig J2 ; VANDERBILT, Colleen A1 ; MCGLYNN, Kathleen1 ; ARNETTE, Don1 ; WEI CHEN1 ; COLLINS, Julie1 ; NAZIRUDDIN, Bashoo3 ; LEVY, Marlon F3
[1] Department of Pharmacology, University of Texas Southwestern Medical Center at Dallas, Dallas, Texas, United States
[2] Departments of Pharmacology and Cellular and Molecular Physiology, Yale University, New Haven, Connecticut, United States
[3] cGMP Islet Cell Processing Laboratory, Islet Cell Transplant Program, Baylor University Medical Center, Dallas, Texas, United States
Source

Diabetes (New York, NY). 2006, Vol 55, Num 4, pp 1066-1073, 8 p ; ref : 47 ref

CODEN
DIAEAZ
ISSN
0012-1797
Scientific domain
Endocrinology; Nutrition, obesity, metabolic disorders
Publisher
American Diabetes Association, Alexandria, VA
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Adrénaline Cellule β Diabète Extracellular signal-regulated protein kinase Glucose Inhibition Catécholamine Endocrinopathie Enzyme Ilot Langerhans Pancréas endocrine
Keyword (en)
Epinephrine β Cell Diabetes mellitus Extracellular signal-regulated protein kinase Glucose Inhibition Catecholamine Endocrinopathy Enzyme Langerhans islet Endocrine pancreas
Keyword (es)
Adrenalina Célula β Diabetes Extracellular signal-regulated protein kinase Glucosa Inhibición Catecolamina Endocrinopatía Enzima Isla Langerhans Páncreas endocrino
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B21 Endocrinopathies / 002B21E Endocrine pancreas. Apud cells (diseases) / 002B21E01 Diabetes. Impaired glucose tolerance / 002B21E01A Etiopathogenesis. Screening. Investigations. Target tissue resistance

Discipline
Endocrinopathies
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
17650563

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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