Developmental damage, increased lipid peroxidation, diminished cyclooxygenase-2 gene expression, and lowered prostaglandin E2 levels in rat embryos exposed to a diabetic environment
- Auteur
- WENTZEL, P1 ; WELSH, N1 ; ERIKSSON, U. J1
[1] Department of Medical Cell Biology, University of Uppsala, Biomedicum, Uppsala, Sweden - Source
Diabetes (New York, NY). 1999, Vol 48, Num 4, pp 813-820 ; ref : 57 ref
- CODEN
- DIAEAZ
- ISSN
- 0012-1797
- Domaine scientifique
- Endocrinology; Nutrition, obesity, metabolic disorders
- Editeur
- American Diabetes Association, Alexandria, VA
- Pays de publication
- United States
- Type de document
- Article
- Langue
- English
- Mot clé (fr)
- Animal Diabète Développement embryonnaire Embryon Expression génique Gestation Mère pathologie Peroxydation Prostaglandin-endoperoxide synthase Prostaglandine E2 Rat Arachidonique acide dérivé Eicosanoïde Embryopathie Endocrinopathie Enzyme Mammalia Oxidoreductases Rodentia Stress oxydatif Vertebrata
- Mot clé (en)
- Animal Diabetes mellitus Embryonic development Embryo Gene expression Pregnancy Maternal diseases Peroxidation Prostaglandin-endoperoxide synthase Prostaglandin E2 Rat Arachidonic acid derivatives Eicosanoid Embryopathy Endocrinopathy Enzyme Mammalia Oxidoreductases Rodentia Oxidative stress Vertebrata
- Mot clé (es)
- Animal Diabetes Desarrollo embrionario Embrión Expresión genética Gestación Madre patología Peroxidación Prostaglandin-endoperoxide synthase Prostaglandina E2 Rata Araquidónico ácido derivado Eicosanoide Embriopatía Endocrinopatía Enzima Mammalia Oxidoreductases Rodentia Stress oxidativo Vertebrata
- Classification
- Pascal
- 002 Sciences biologiques et medicales / 002B Sciences medicales / 002B21 Endocrinopathies / 002B21E Pancréas endocrine. Cellules apud (pathologie) / 002B21E01 Diabète. Anomalie tolérance glucose / 002B21E01A Etiopathogénie. Dépistage. Explorations. Résistance tissu cible
- Discipline
- Endocrinopathies
- Provenance
- Inist-CNRS
- Base de données
- PASCAL
- Identifiant INIST
- 1776099
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.2337/diabetes.48.4.813 
https://dx.doi.org/10.2337/diabetes.48.4.813
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