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Inactivation of misselected CD8 T cells by CD8 gene methylation and cell death

Author
PESTANO, G. A1 2 ; YALING ZHOU1 2 ; TRIMBLE, L. A1 2 ; DALEY, J3 ; WEBER, G. F1 4 ; CANTOR, H1 2
[1] Department of Cancer Immunology and AIDS, Dana Farber Cancer Institute, United States
[2] Department of Pathology, Harvard Medical School, 44 Binney Street, Boston MA 02115, United States
[3] Department of Adult Oncology, Dana Farber Cancer Institute, United States
[4] Department of Medicine, Harvard Medical School, 44 Binney Street, Boston MA 02115, United States
Source

Science (Washington, D.C.). 1999, Vol 284, Num 5417, pp 1187-1191 ; ref : 32 ref

CODEN
SCIEAS
ISSN
0036-8075
Scientific domain
Multidisciplinary
Publisher
American Association for the Advancement of Science, Washington, DC
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Apoptose Développement Expression génique Gène Inactivation Lymphocyte T Méthylation Récepteur cellule T Régulation Répertoire immunologique Souris Thymocyte Antigène CD8 Mammalia Rodentia Vertebrata
Keyword (en)
Apoptosis Development Gene expression Gene Inactivation T-Lymphocyte Methylation T cell receptor Regulation(control) Immunologic repertoire Mouse Thymocyte Mammalia Rodentia Vertebrata
Keyword (es)
Apoptosis Desarrollo Expresión genética Gen Inactivación Linfocito T Metilación Receptor célula T Regulación Repertorio inmunologico Ratón Timocito Mammalia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A06 Fundamental immunology / 002A06C Immunobiology / 002A06C02 Lymphoid organs: ontogeny, organization, homing phenomenon

Discipline
Fundamental immunology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
1788132

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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