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Inhibitory effect of Shiga toxin on the expression of Na+-D-glucose cotransporter in rabbit ileum

Author
CHOPRA, Puja1 ; VERMA, Dinesh1 ; KHULLAR, Madhu1 ; MAHMOOD, Safrun1
[1] Department of Experimental Medicine and Biotechnology, Postgraduate Institute of Medical Education and Research, Chandigarh- 160012, India
Source

Nutrition research (New York, NY). 2006, Vol 26, Num 7, pp 356-361, 6 p ; ref : 23 ref

CODEN
NTRSDC
ISSN
0271-5317
Scientific domain
Nutrition, obesity, metabolic disorders
Publisher
Elsevier Science, New York, NY
Publication country
United States
Document type
Article
Language
English
Author keyword
Rabbit ileum Shiga toxin Shigella dysenteriae 1 Sodium-dependent glucose transporter (SGLT1) protein expression mRNA
Keyword (fr)
Animal Expression génique Glucose Iléon Lapin Mammalia Protéine transport RNA messager Sodium Toxine Transport couplé Appareil digestif Intestin grêle Lagomorpha Vertebrata
Keyword (en)
Animal Gene expression Glucose Ileum Rabbit Mammalia Carrier protein Messenger RNA Sodium Toxin Coupled transport Digestive system Small intestine Lagomorpha Vertebrata
Keyword (es)
Animal Expresión genética Glucosa Ileon Conejo Mammalia Proteína transportador RNA mensajero Sodio Toxina Transporte acoplado Aparato digestivo Intestino delgado Lagomorpha Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A16 Vertebrates: anatomy and physiology, studies on body, several organs or systems / 002A16E Feeding. Feeding behavior

Discipline
Vertebrates : anatomy and physiology, studies on body, several organs or systems
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
18107644

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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