Inhibition of HtrA2/Omi ameliorates heart dysfunction following ischemia/ reperfusion injury in rat heart in vivo
- Author
- SHENUARIN BHUIYAN, Md1 ; FUKUNAGA, Kohji1 2
[1] Department of Pharmacology, Graduate School of Pharmaceutical Sciences, Tohoku University, Sendai, Japan
[2] Tohoku University 21st Century COE Program CRESCENDO, Sendai, Japan - Source
European journal of pharmacology. 2007, Vol 557, Num 2-3, pp 168-177, 10 p ; ref : 1 p.1/4
- CODEN
- EJPHAZ
- ISSN
- 0014-2999
- Scientific domain
- Pharmacology drugs
- Publisher
- Elsevier, Amsterdam
- Publication country
- Netherlands
- Document type
- Article
- Language
- English
- Author keyword
- Cardioprotection HtrA2 (High temperature requirement A2) Ischemia/reperfusion UCF-101 XIAP (X-linked inhibitor of apoptosis protein)
- Keyword (fr)
- Animal Apoptose Cardioprotecteur Coeur In vivo Inhibiteur protéique Ischémie reperfusion Mort cellulaire Rat Température Trouble fonctionnel Appareil circulatoire Mammalia Rodentia Vertebrata
- Keyword (en)
- Animal Apoptosis Cardioprotective agent Heart In vivo Protein inhibitor Ischemia reperfusion Cell death Rat Temperature Dysfunction Circulatory system Mammalia Rodentia Vertebrata
- Keyword (es)
- Animal Apoptosis Cardioprotector Corazón In vivo Inhibidor proteico Isquemia reperfusión Muerte celular Rata Temperatura Trastorno funcional Aparato circulatorio Mammalia Rodentia Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B12 Cardiology. Vascular system
- Discipline
- Cardiology. Circulatory system General pharmacology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 18497281
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1016/j.ejphar.2006.10.067 
https://dx.doi.org/10.1016/j.ejphar.2006.10.067
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