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Differentially androgen-modulated genes in ovarian epithelial cells from BRCA mutation carriers and control patients predict ovarian cancer survival and disease progression

Author
MOTAMED -KHORASANI, A1 2 ; JURISICA, I3 4 5 ; LETARTE, M5 6 7 8 ; SHAW, P. A7 9 ; PARKES, R. K1 ; ZHANG, X3 ; EVANGELOU, A1 6 ; ROSEN, B10 7 ; MURPHY, K. J10 7 ; BROWN, T. J1 2 7
[1] The Samuel Lunenfeld Research Institute, Mt Sinai Hospital, Toronto, Ontario, Canada
[2] Department ofPhysiology, University of Toronto, Toronto, Ontario, Canada
[3] Division of Signaling Biology, Ontario Cancer Institute/Princess Margaret Hospital, University Health Network, Toronto, Ontario, Canada
[4] Department of Computer Science, University of Toronto, Toronto, Ontario, Canada
[5] Department of Medical Biophysics, University of Toronto, Toronto, Ontario, Canada
[6] Cancer Research Program, The Hospital for Sick Children, University of Toronto, Toronto, Ontario, Canada
[7] Department of Obstetrics and Gynecology, University of Toronto, Toronto, Ontario, Canada
[8] Department of Immunology, University of Toronto, Toronto, Ontario, Canada
[9] Division of Pathology, Ontario Cancer Institute/Princess Margaret Hospital, University Health Network, University of Toronto, Toronto, Ontario, Canada
[10] Division of Gynecologic Oncology, Ontario Cancer Institute/Princess Margaret Hospital, University Health Network, University of Toronto, Toronto, Ontario, Canada
Source

Oncogene (Basingstoke). 2007, Vol 26, Num 2, pp 198-214, 17 p ; ref : 1 p

ISSN
0950-9232
Scientific domain
Cell biology, histology; Medical oncology; Genetics
Publisher
Nature Publishing, Basingstoke
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
BACH2 BRCA1 BRCA2 acetylcholinesterase androgen ovarian cancer
Keyword (fr)
Acetylcholinesterase Androgène Cancer ovaire Carcinogenèse Cellule épithéliale Gène BRCA1 Gène BRCA2 Gène suppresseur tumeur Homme Mutation Porteur Régulation Survie Appareil génital femelle pathologie Carboxylic ester hydrolases Enzyme Esterases Hormone stéroïde sexuelle Hydrolases Ovaire pathologie Tumeur maligne
Keyword (en)
Acetylcholinesterase Androgen Ovary cancer Carcinogenesis Epithelial cell BRCA1 gene BRCA2 gene Tumor suppressor gene Human Mutation Carrier Regulation(control) Survival Female genital diseases Carboxylic ester hydrolases Enzyme Esterases Sex steroid hormone Hydrolases Ovarian diseases Malignant tumor
Keyword (es)
Acetylcholinesterase Andrógeno Cáncer del ovario Carcinogénesis Célula epitelial Gen BRCA1 Gen BRCA2 Gen supresor tumor Hombre Mutación Portador Regulación Sobrevivencia Aparato genital hembra patología Carboxylic ester hydrolases Enzima Esterases Hormona esteroide sexual Hydrolases Ovario patología Tumor maligno
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes

Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B20 Gynecology. Andrology. Obstetrics / 002B20C Female genital diseases / 002B20C02 Tumors

Discipline
Gynecology. Andrology. Obstetrics Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
18521882

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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