Disrupting the pairing between let-7 and Hmga2 enhances oncogenic transformation
- Author
- MAYR, Christine1 ; HEMANN, Michael T2 ; BARTEL, David P1
[1] Howard Hughes Medical Institute and Department of Biology, Massachusetts Institute of Technology, and Whitehead Institute for Biomedical Research, 9 Cambridge Center, Cambridge, MA 02142, United States
[2] Center for Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139, United States - Source
Science (Washington, D.C.). 2007, Vol 315, Num 5818, pp 1576-1579, 4 p ; ref : 29 ref
- CODEN
- SCIEAS
- ISSN
- 0036-8075
- Scientific domain
- Multidisciplinary
- Publisher
- American Association for the Advancement of Science, Washington, DC
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Carcinogenèse Expression génique Répression Souris Gène Hmga2 Micro ARN Mammalia Rodentia Silence expression génique Vertebrata
- Keyword (en)
- Carcinogenesis Gene expression Repression Mouse Micro RNA Mammalia Rodentia Gene silencing Vertebrata
- Keyword (es)
- Carcinogénesis Expresión genética Represión Ratón Micro ARN Mammalia Rodentia Silencio expresión genética Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes
- Discipline
- Molecular and cell biology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 18625440
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1126/science.1137999 
https://dx.doi.org/10.1126/science.1137999
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