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High glucose impairs early and late endothelial progenitor cells by modifying nitric oxide-related but not oxidative stress-mediated mechanisms

Author
CHEN, Yung-Hsiang1 2 3 ; LIN, Shing-Jong1 2 4 ; LIN, Feng-Yen1 4 ; WU, Tao-Cheng1 2 4 ; TSAO, Chen-Rong1 5 ; HUANG, Po-Hsun1 2 4 ; LIU, Po-Len1 6 ; CHEN, Yuh-Lien7 ; CHEN, Jaw-Wen1 2 4
[1] School of Medicine, National Yang-Ming University, Taipei City, Taiwan, Province of China
[2] Cardiovascular Research Center, National Yang-Ming University, Taipei City, Taiwan, Province of China
[3] Graduate Institute of Integrated Medicine, China Medical University, Taichung, Taiwan, Province of China
[4] Division of Cardiology, Department of Medicine, National Taipei Veterans General Hospital, Taipei City, Taiwan, Province of China
[5] Taichung Veterans General Hospital, Taichung, Taiwan, Province of China
[6] Faculty of Respiratory Care, Kaohsiung Medical University, Kaohsiung, Taiwan, Province of China
[7] Department of Anatomy and Cell Biology, National Taiwan University, Taipei, Taiwan, Province of China
Source

Diabetes (New York, NY). 2007, Vol 56, Num 6, pp 1559-1568, 10 p ; ref : 50 ref

CODEN
DIAEAZ
ISSN
0012-1797
Scientific domain
Endocrinology; Nutrition, obesity, metabolic disorders
Publisher
American Diabetes Association, Alexandria, VA
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Azote monoxyde Cellule endothéliale Cellule précurseur Diabète Glucose Stress oxydatif Endocrinopathie
Keyword (en)
Nitric oxide Endothelial cell Precursor cell Diabetes mellitus Glucose Oxidative stress Endocrinopathy
Keyword (es)
Nitrógeno monóxido Célula endotelial Célula precursor Diabetes Glucosa Estrés oxidativo Endocrinopatía
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B21 Endocrinopathies / 002B21E Endocrine pancreas. Apud cells (diseases) / 002B21E01 Diabetes. Impaired glucose tolerance / 002B21E01A Etiopathogenesis. Screening. Investigations. Target tissue resistance

Discipline
Endocrinopathies
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
18811661

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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