ABT-737, an inhibitor of Bcl-2 family proteins, is a potent inducer of apoptosis in multiple myeloma cells
- Author
- KLINE, M. P1 ; RAJKUMAR, S. V1 ; TIMM, M. M1 ; KIMLINGER, T. K1 ; HAUG, J. L1 ; LUST, J. A1 ; GREIPP, P. R1 ; KUMAR, S1
[1] Division of Hematology, Department of Internal Medicine, Mayo Clinic, Rochester, MN, United States - Source
Leukemia. 2007, Vol 21, Num 7, pp 1549-1560, 12 p ; ref : 40 ref
- CODEN
- LEUKED
- ISSN
- 0887-6924
- Scientific domain
- Medical oncology; Hematology
- Publisher
- Nature Publishing, London
- Publication country
- United Kingdom
- Document type
- Article
- Language
- English
- Author keyword
- Bcl-2 Bcl-xL apoptosis multiple myeloma
- Keyword (fr)
- Anticancéreux Apoptose Cancérologie Gène onc cellulaire Homme Hématologie Inhibiteur protéique Mort cellulaire Myélome Protooncogène Traitement Gène BclxL Gène bcl2 Génétique Hémopathie maligne Immunoglobulinopathie Immunopathologie Lymphoprolifératif syndrome
- Keyword (en)
- Antineoplastic agent Apoptosis Cancerology C-Onc gene Human Hematology Protein inhibitor Cell death Myeloma Protooncogene Treatment Genetics Malignant hemopathy Immunoglobulinopathy Immunopathology Lymphoproliferative syndrome
- Keyword (es)
- Anticanceroso Apoptosis Cancerología Gen onc celular Hombre Hematología Inhibidor proteico Muerte celular Mieloma Protooncogen Tratamiento Genética Hemopatía maligna Inmunoglobulinopatía Inmunopatología Linfoproliferativo síndrome
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments / 002B02R Antineoplastic agents / 002B02R01 General aspects
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B06 Immunopathology / 002B06D Immunodeficiencies. Immunoglobulinopathies / 002B06D02 Immunoglobulinopathies
- Discipline
- Immunopathology Pharmacological treatments
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 18867660
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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