Increased wnt signaling during aging alters muscle stem cell fate and increases fibrosis
- Author
- BRACK, Andrew S1 ; CONBOY, Michael J1 ; ROY, Sudeep1 ; LEE, Mark2 ; KUO, Calvin J2 ; KELLER, Charles3 ; RANDO, Thomas A1 4
[1] Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA 94305, United States
[2] Department of Medicine, Division of Hematology, Stanford University School of Medicine, Stanford, CA 94305, United States
[3] Department of Cellular and Structural Biology, The University of Texas Health Science Center, San Antonio, TX 78229, United States
[4] Geriatric Research, Education, and Clinical Center (GRECC) and Neurology Service, Veterans Affairs (VA) Palo Alto Health Care System, Palo Alto, CA 94304, United States - Source
Science (Washington, D.C.). 2007, Vol 317, Num 5839, pp 807-810, 4 p
- CODEN
- SCIEAS
- ISSN
- 0036-8075
- Scientific domain
- Multidisciplinary
- Publisher
- American Association for the Advancement of Science, Washington, DC
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Animal âgé Cellule satellite Cellule souche Fibrose Modèle animal Muscle strié Promoteur transcription Souris Sénescence Transduction signal Vieillissement Mammalia Rodentia Vertebrata
- Keyword (en)
- Aged animal Satellite cell Stem cell Fibrosis Animal model Striated muscle Transcription promoter Mouse Senescence Signal transduction Ageing Mammalia Rodentia Vertebrata
- Keyword (es)
- Animal viejo Célula satélite Célula primitiva Fibrosis Modelo animal Músculo estriado Promotor transcripción Ratón Senescencia Transducción señal Envejecimiento Mammalia Rodentia Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A23 Vertebrates: osteoarticular system, musculoskeletal system / 002A23B Striated muscle. Tendons
- Discipline
- Vertebrates : osteoarticular system, musculoskeletal system
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 18996775
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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