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Induction of mitochondria-involved apoptosis in estrogen receptor-negative cells by a novel tamoxifen derivative, ridaifen-B

Author
NAGAHARA, Yukitoshi1 ; SHIINA, Isamu2 ; NAKATA, Kenya2 ; SASAKI, Akane2 ; MIYAMOTO, Tomomi2 ; IKEKITA, Masahiko3
[1] Department of Biotechnology, College of Science and Engineering, Tokyo Denki University, Hatoyama, Hiki-gun, Saitama, 350-0394, Japan
[2] Department of Applied Chemistry, Faculty of Science, Tokyo University of Science, 1-3 Kagurazaka, Shinjuku-ku, Tokyo 162-8601, Japan
[3] Department of Applied Biological Science, Faculty of Science and Technology, Tokyo University of Science, 2641 Yamazaki, Noda, Chiba 278-8510, Japan
Source

Cancer science. 2008, Vol 99, Num 3, pp 608-614, 7 p ; ref : 26 ref

ISSN
1347-9032
Scientific domain
Medical oncology
Publisher
Blackwell, Tokyo
Publication country
Japan
Document type
Article
Language
English
Keyword (fr)
Anticancéreux Apoptose Cancérologie Mitochondrie Mort cellulaire Récepteur hormonal Récepteur oestrogène Tamoxifène Antihormone Antioestrogène Composé non stéroïde Modulateur sélectif des récepteurs aux oestrogènes
Keyword (en)
Antineoplastic agent Apoptosis Cancerology Mitochondria Cell death Hormonal receptor Estrogen receptor Tamoxifene Antihormone Antiestrogen Non steroid compound Selective estrogen receptor modulator
Keyword (es)
Anticanceroso Apoptosis Cancerología Mitocondria Muerte celular Receptor hormonal Receptor estrógeno Tamoxifeno Antihormona Antiestrógeno Compuesto no esteroide Modulador selectivo del receptor de estrógeno
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B04 Tumors

Discipline
Tumours
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
20122500

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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