A selective phosphodiesterase 4 (PDE4) inhibitor Zl-n-91 suppresses IL-17 production by human memory Th17 cells
- Author
- RUIHONG MA1 ; YANG, Bin-Yan1 ; WU, Chang-You1
[1] Department of Immunology, Zhongshan School of Medicine, Sun Yat-sen University, Key Laboratory of Tropical Diseases Control, Guangzhou 510080, China - Source
International immunopharmacology. 2008, Vol 8, Num 10, pp 1408-1417, 10 p ; ref : 37 ref
- ISSN
- 1567-5769
- Scientific domain
- Immunology, immunopathology, allergology; Pharmacology drugs
- Publisher
- Elsevier, Amsterdam
- Publication country
- Netherlands
- Document type
- Article
- Language
- English
- Author keyword
- Autoimmune diseases Memory T cells PDE4 Th17
- Keyword (fr)
- Biosynthèse Cellule helper Homme Inhibiteur de la phosphodiesterase Inhibiteur de la phosphodiestérase 4 Interleukine 17 Lymphocyte T Maladie autoimmune Production Lymphocyte Th17 Lymphocyte mémoire Immunopathologie
- Keyword (en)
- Biosynthesis Helper cell Human Phosphodiesterase inhibitor Phosphodiesterase 4 inhibitor Interleukin 17 T-Lymphocyte Autoimmune disease Production Th17 lymphocyte Memory lymphocyte Immunopathology
- Keyword (es)
- Biosíntesis Célula auxiliar Hombre Inhibidor phosphodiesterase Inhibidor fosfodiesterasa 4 Interleuquina 17 Linfocito T Enfermedad autoinmune Producción Linfocito TH17 Inmunopatología
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments
- Discipline
- General pharmacology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 20616189
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1016/j.intimp.2008.05.012 
https://dx.doi.org/10.1016/j.intimp.2008.05.012
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