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Suppression of transient receptor potential melastatin 7 channel induces cell death in gastric cancer

Author
BYUNG JOO KIM1 ; EUN JUNG PARK1 ; JAE HWA LEE1 ; JEON, Ju-Hong1 ; SEON JEONG KIM2 ; SO, Insuk1
[1] Center for Bio-Artificial Muscle and Department of Physiology, Seoul National University College of Medicine, Seoul 110-799, Korea, Republic of
[2] Center for Bio-Artificial Muscle and Department of Biomedical Engineering, Hanyang University, Seoul 133-791, Korea, Republic of
Source

Cancer science. 2008, Vol 99, Num 12, pp 2502-2509, 8 p ; ref : 56 ref

ISSN
1347-9032
Scientific domain
Medical oncology
Publisher
Blackwell, Richmond
Publication country
Australia
Document type
Article
Language
English
Keyword (fr)
Apoptose Canal ionique Cancer de l'estomac Cancérologie Cellule tumorale Mort cellulaire Récepteur biologique Transitoire Cancer Pathologie de l'appareil digestif Pathologie de l'estomac Tumeur maligne
Keyword (en)
Apoptosis Ionic channel Stomach cancer Cancerology Tumor cell Cell death Biological receptor Transitory Cancer Digestive diseases Gastric disease Malignant tumor
Keyword (es)
Apoptosis Canal iónico Cáncer del estómago Cancerología Célula tumoral Muerte celular Receptor biológico Transitorio Cáncer Aparato digestivo patología Estómago patología Tumor maligno
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B04 Tumors

Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B13 Gastroenterology. Liver. Pancreas. Abdomen / 002B13B Stomach. Duodenum. Small intestine. Colon. Rectum. Anus / 002B13B01 Tumors

Discipline
Gastroenterology. Liver. Pancreas. Abdomen Tumours
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
20954936

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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