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CD24 and Siglec-10 Selectively Repress Tissue Damage-Induced Immune Responses

Author
CHEN, Guo-Yun1 ; JIE TANG2 ; PAN ZHENG1 3 ; YANG LIU1 4
[1] Division of Immunotherapy, Department of Surgery, University of Michigan School of Medicine, Ann Arbor, MI 48109, United States
[2] Institute of Biophysics, Chinese Academy of Science, Beijing, China
[3] Department of Pathology, University of Michigan School of Medicine, Ann Arbor, MI 48109, United States
[4] Department of Internal Medicine, University of Michigan School of Medicine, Ann Arbor, MI 48109, United States
Source

Science (Washington, D.C.). 2009, Vol 323, Num 5922, pp 1722-1725, 4 p ; ref : 29 ref

CODEN
SCIEAS
ISSN
0036-8075
Scientific domain
Multidisciplinary
Publisher
American Association for the Advancement of Science, Washington, DC
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Immunité naturelle Lésion Régulation Réponse immune Répression Souris Tissu Gène Cd24a Gène Siglecg Motif moléculaire associé au danger Motif moléculaire associé aux pathogènes Récepteur PRR Mammalia Rodentia Vertebrata
Keyword (en)
Natural immunity Lesion Regulation(control) Immune response Repression Mouse Tissue Danger-associated molecular pattern Pathogen-associated molecular pattern PRR receptor Mammalia Rodentia Vertebrata
Keyword (es)
Inmunidad natural Lesión Regulación Respuesta inmune Represión Ratón Tejido Mammalia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A06 Fundamental immunology / 002A06C Immunobiology / 002A06C06 Genetics of the immune response

Discipline
Fundamental immunology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
21358522

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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