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Doxycycline reduces mortality to lethal endotoxemia by reducing nitric oxide synthesis via an interleukin-10-independent mechanism

Author
D'AGOSTINO, P1 ; LA ROSA, M1 ; BARBERA, C1 ; ARCOLEO, F1 ; DI BELLA, G1 ; MILANO, S1 ; CILLARI, E1
[1] Institute of General Pathology, University of Palermo, Italy
Source

The Journal of infectious diseases. 1998, Vol 177, Num 2, pp 489-492 ; ref : 14 ref

CODEN
JIDIAQ
ISSN
0022-1899
Scientific domain
Immunology, immunopathology, allergology; Microbiology, infectious diseases; Tropical medicine
Publisher
University of Chicago Press, Chicago, IL
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Activité biologique Antiinflammatoire Azote monoxyde Choc Doxycycline Endotoxémie Inhibition Libération Lipopolyoside Mécanisme Nitrate Septicémie Tétracycline dérivé Appareil circulatoire pathologie
Keyword (en)
Biological activity Antiinflammatory agent Nitric oxide Shock Doxycycline Endotoxemia Inhibition Release Lipopolysaccharide Mechanism Nitrates Septicemia Tetracycline derivatives Cardiovascular disease
Keyword (es)
Actividad biológica Antiinflamatorio Nitrógeno monóxido Choque Doxiciclina Endotoxemia Inhibición Liberación Lipopoliósido Mecanismo Nitrato Septicemia Tetraciclina derivado Aparato circulatorio patología
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments / 002B02L Bones, joints and connective tissue. Antiinflammatory agents

Discipline
Pharmacological treatments
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
2148525

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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