Hyperglycemia Impairs Proteasome Function by Methylglyoxal
- Autor
- QUEISSER, Markus A1 2 ; DACHUN YAO2 ; GEISLER, Sven1 ; HAMMES, Hans-Peter3 ; LOCHNIT, Günter1 ; SCHLEICHER, Erwin D4 5 ; BROWNLEE, Michael2 ; PREISSNER, Klaus T1
[1] School of Medicine, Institute of Biochemistry, Justus-Liebig-University, Giessen, Germany
[2] Diabetes Center, Albert Einstein College of Medicine, New York, New York, United States
[3] Department of Endocrinology, University of Heidelberg, Mannheim, Germany
[4] Department of Medicine IV, Eberhard-Karls-University, Tübingen, Germany
[5] Department of Neurology, Eberhard-Karls-University, Tübingen, Germany - Fuente
Diabetes (New York, NY). 2010, Vol 59, Num 3, pp 670-678, 9 p ; ref : 39 ref
- CODEN
- DIAEAZ
- ISSN
- 0012-1797
- Campo Científico
- Endocrinology; Nutrition, obesity, metabolic disorders
- Editor
- American Diabetes Association, Alexandria, VA
- País de la publicación
- United States
- Tipo de documento
- Article
- Idioma
- English
- Palabra clave (fr)
- Diabète Hyperglycémie Endocrinopathie
- Palabra clave (in)
- Diabetes mellitus Hyperglycemia Endocrinopathy
- Palabra clave (es)
- Diabetes Hiperglicemia Endocrinopatía
- Clasificación
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B21 Endocrinopathies / 002B21E Endocrine pancreas. Apud cells (diseases) / 002B21E01 Diabetes. Impaired glucose tolerance / 002B21E01A Etiopathogenesis. Screening. Investigations. Target tissue resistance
- Disciplina
- Endocrinopathies
- Procedencia
- Inist-CNRS
- Base de datos
- PASCAL
- Identificador INIST
- 22505339
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.2337/db08-1565 
https://dx.doi.org/10.2337/db08-1565
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