DNA glycosylase encoded by MUTYH functions as a molecular switch for programmed cell death under oxidative stress to suppress tumorigenesis
- Author
- OKA, Sugako1 ; NAKABEPPU, Yusaku1
[1] Division of Neurofunctional Genomics, Department of Immunobiology and Neuroscience, Medical Institute of Bioregulation, Kyushu University, Fukuoka, Japan - Source
Cancer science. 2011, Vol 102, Num 4, pp 677-682, 6 p ; ref : 49 ref
- ISSN
- 1347-9032
- Scientific domain
- Medical oncology
- Publisher
- Blackwell, Richmond
- Publication country
- Australia
- Document type
- Article
- Language
- English
- Keyword (fr)
- Apoptose Cancérologie Carcinogenèse DNA Glycosylases Gène réparation Mort cellulaire Stress oxydatif Tumorigénicité Gène MYH Enzyme Hydrolases
- Keyword (en)
- Apoptosis Cancerology Carcinogenesis DNA Glycosylases Repair gene Cell death Oxidative stress Tumorigenicity Enzyme Hydrolases
- Keyword (es)
- Apoptosis Cancerología Carcinogénesis DNA Glycosylases Gen reparación Muerte celular Estrés oxidativo Tumorigenicidad Enzima Hydrolases
- Classification
- Discipline
- Tumours
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 24085986
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1111/j.1349-7006.2011.01869.x 
https://dx.doi.org/10.1111/j.1349-7006.2011.01869.x
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