Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis
- Author
- International Multiple Sclerosis Genetics Consortium
- Source
Nature (London). 2011, Vol 476, Num 7359, pp 214-219, 6 p ; ref : 26 ref
- CODEN
- NATUAS
- ISSN
- 0028-0836
- Scientific domain
- Multidisciplinary
- Publisher
- Nature Publishing Group, London
- Publication country
- United Kingdom
- Document type
- Article
- Language
- English
- Keyword (fr)
- Génétique Locus HLA-A Pathogénie Risque Sclérose en plaques Association génétique Echelle du génome Locus HLA-DRB1 Europe Maladie inflammatoire Pathologie du système nerveux central Pathologie du système nerveux
- Keyword (en)
- Genetics HLA-A-Locus Pathogenesis Risk Multiple sclerosis Europe Inflammatory disease Central nervous system disease Nervous system diseases
- Keyword (es)
- Genética Locus HLA-A Patogenia Riesgo Esclerosis en placa Europa Enfermedad inflamatoria Sistema nervosio central patología Sistema nervioso patología
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A07 Genetics of eukaryotes. Biological and molecular evolution / 002A07E Population genetics, reproduction patterns / 002A07E03 Human
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B17 Neurology / 002B17F Multiple sclerosis and variants. Guillain barré syndrome and other inflammatory polyneuropathies. Leukoencephalitis
- Discipline
- Eukaryotes genetics. Biological and molecular evolution Neurology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 24404342
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1038/nature10251 
https://dx.doi.org/10.1038/nature10251
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