Inhibition of inflammation by pentosan polysulfate impedes the development and progression of severe diabetic nephropathy in aging C57B6 mice
- Author
- JIN WU1 ; GUAN, Tian-Jun2 ; SHIRONG ZHENG3 ; GROSJEAN, Fabrizio1 ; WEICHENG LIU4 ; HUABAO XIONG4 ; GORDON, Ronald5 ; VLASSARA, Helen1 ; STRIKER, Gary E1 6 ; FENG ZHENG1 7
[1] Division of Experimental Diabetes and Aging, Department of Geriatrics, Mount Sinai School of Medicine, New York, NY, United States
[2] Department of Nephrology, Zhongshan Hospital, Xiamen University School of Medicine, Xiamen, Fujian, China
[3] Department of Pediatrics, University of Louisville, Louisville, KY, United States
[4] Immunobiology Institute, Mount Sinai School of Medicine, New York, NY, United States
[5] Department of Pathology, Mount Sinai School of Medicine, New York, NY, United States
[6] Division of Nephrology, Department of Medicine, Mount Sinai School of Medicine, New York, NY, United States
[7] Department of Nephrology, Dongfang Hospital, Xiamen University, Fuzhou, Fujian, China - Source
Laboratory investigation. 2011, Vol 91, Num 10, pp 1459-1471, 13 p ; ref : 50 ref
- CODEN
- LAINAW
- ISSN
- 0023-6837
- Scientific domain
- Biochemistry, molecular biology, biophysics; Medical laboratory techniques; General medicine general surgery
- Publisher
- Nature Publishing Group, New York, NY
- Publication country
- United States
- Document type
- Article
- Language
- English
- Author keyword
- PPS TNFα aging diabetic nephropathy inflammation
- Keyword (fr)
- Age Animal Anticoagulant Biologie clinique Biotechnologie Diabète Développement Evolution Facteur nécrose tumorale α Grave Inflammation Inhibiteur Inhibition Néphropathie diabétique Pentosane polysulfate sodique Pronostic Souris Sénescence Vieillissement Association morbide Cytokine Endocrinopathie Mammalia Pathologie de l'appareil urinaire Pathologie du rein Rodentia Vertebrata
- Keyword (en)
- Age Animal Anticoagulant Clinical biology Biotechnology Diabetes mellitus Development Evolution Tumor necrosis factor α Severe Inflammation Inhibitor Inhibition Diabetic nephropathy Pentosan polysulfate sodium Prognosis Mouse Senescence Ageing Concomitant disease Cytokine Endocrinopathy Mammalia Urinary system disease Kidney disease Rodentia Vertebrata
- Keyword (es)
- Edad Animal Anticoagulante Biología clínica Biotecnología Diabetes Desarrollo Evolución Factor necrosis tumoral α Grave Inflamación Inhibidor Inhibición Nefropatía diabética Pentosano polisulfato de sodio Pronóstico Ratón Senescencia Envejecimiento Asociación morbosa Citoquina Endocrinopatía Mammalia Aparato urinario patología Riñón patología Rodentia Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A31 Biotechnology
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B21 Endocrinopathies / 002B21E Endocrine pancreas. Apud cells (diseases) / 002B21E01 Diabetes. Impaired glucose tolerance / 002B21E01A Etiopathogenesis. Screening. Investigations. Target tissue resistance
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B24 Investigative techniques, diagnostic techniques (general aspects)
- Discipline
- Biotechnology Endocrinopathies Scanning and diagnostic techniques
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 24587227
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1038/labinvest.2011.93 
https://dx.doi.org/10.1038/labinvest.2011.93
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