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PTPN2, a Candidate Gene for Type 1 Diabetes, Modulates Pancreatic β-Cell Apoptosis via Regulation of the BH3-Only Protein Bim

Author
SANTIN, Izortze1 ; MOORE, Fabrice1 ; COLLI, Maikel L1 ; GURZOV, Esteban N1 ; MARSELLI, Lorella2 ; MARCHETTI, Piero2 ; EIZIRIK, Decio L1
[1] Laboratory of Experimental Medicine, Université Libre de Bruxelles, Brussels, Belgium
[2] Department of Endocrinology and Metabolism, Metabolic Unit, University of Pisa, Pisa, Italy
Source

Diabetes (New York, NY). 2011, Vol 60, Num 12, pp 3279-3288, 10 p ; ref : 50 ref

CODEN
DIAEAZ
ISSN
0012-1797
Scientific domain
Endocrinology; Nutrition, obesity, metabolic disorders
Publisher
American Diabetes Association, Alexandria, VA
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Apoptose Cellule β Diabète de type 1 Gène candidat Protéine Endocrinopathie Ilot Langerhans Immunopathologie Maladie autoimmune Mort cellulaire Pancréas endocrine
Keyword (en)
Apoptosis β Cell Type 1 diabetes Candidate gene Protein Endocrinopathy Langerhans islet Immunopathology Autoimmune disease Cell death Endocrine pancreas
Keyword (es)
Apoptosis Célula β Diabetes de tipo 1 Gen candidato Proteína Endocrinopatía Isla Langerhans Inmunopatología Enfermedad autoinmune Muerte celular Páncreas endocrino
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B21 Endocrinopathies / 002B21E Endocrine pancreas. Apud cells (diseases) / 002B21E01 Diabetes. Impaired glucose tolerance / 002B21E01A Etiopathogenesis. Screening. Investigations. Target tissue resistance

Discipline
Endocrinopathies
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
25273961

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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