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ZAP-70 deficiency in an autosomal recessive form of severe combined immunodeficiency

Author
CHAN, A. C1 ; KADLECEK, T. A1 ; ELDER, M. E; FILIPOVICH, A. H; WEN-LIN KUO; IWASHIMA, M1 ; PARSLOW, T. G; WEISS, A1
[1] Univ. California, Howard Hughe smedical inst., dep. medicine, San Francisco CA 94143, United States
Source

Science (Washington, D.C.). 1994, Vol 264, Num 5165, pp 1599-1601

CODEN
SCIEAS
ISSN
0036-8075
Scientific domain
Multidisciplinary
Publisher
American Association for the Advancement of Science, Washington, DC
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Déficit Etiopathogénie Grave Gène Homme Immunodéficit mixte Lymphocyte T Mutation Protein-tyrosine kinase Récepteur cellule T Transduction signal Enzyme Immunopathologie Maladie héréditaire Transferases
Keyword (en)
Deficiency Etiopathogenesis Severe Gene Human Combined immune deficiency T-Lymphocyte Mutation Protein-tyrosine kinase T cell receptor Signal transduction Enzyme Immunopathology Genetic disease Transferases
Keyword (es)
Déficiencia Etiopatogenia Grave Gen Hombre Inmunodeficiencia mixta Linfocito T Mutación Protein-tyrosine kinase Receptor célula T Transducción señal Enzima Inmunopatología Enfermedad hereditaria Transferases
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B06 Immunopathology / 002B06D Immunodeficiencies. Immunoglobulinopathies / 002B06D01 Immunodeficiencies

Discipline
Immunopathology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
4104763

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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