Amyloid β peptide-induced cerebral endothelial cell death involves mitochondrial dysfunction and caspase activation
- Author
- JAN XU1 ; SHAWEI CHEN1 ; KU, Grace1 ; HINAN AHMED, S1 ; JINMING XU1 ; HONG CHEN1 ; HSU, Chung Y1
[1] Department of Neurology and Center for the Study of Nervous System Injury, Washington University School of Medicine, St. Louis, Missouri, United States - Source
Journal of cerebral blood flow and metabolism. 2001, Vol 21, Num 6, pp 702-710 ; ref : 1 p.1/2
- Publisher
- Lippincott Williams & Wilkins, Hagerstown, MD
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Animal Apoptose Bovin Cellule endothéliale DNA mitochondrial Démence Alzheimer Effet biologique Pathogénie Protéine amyloïde β Souris Stress oxydatif Vaisseau sanguin Caspases Appareil circulatoire Artiodactyla Encéphale pathologie Maladie dégénérative Mammalia Rodentia Système nerveux central pathologie Système nerveux pathologie Ungulata Vertebrata
- Keyword (en)
- Animal Apoptosis Bovine Endothelial cell Mitochondrial DNA Alzheimer disease Biological effect Pathogenesis β Amyloid protein Mouse Oxidative stress Blood vessel Circulatory system Artiodactyla Cerebral disorder Degenerative disease Mammalia Rodentia Central nervous system disease Nervous system diseases Ungulata Vertebrata
- Keyword (es)
- Animal Apoptosis Bovino Célula endotelial DNA mitocondrial Demencia Alzheimer Efecto biológico Patogenia Proteína amiloide β Ratón Stress oxidativo Vaso sanguíneo Aparato circulatorio Artiodactyla Encéfalo patología Enfermedad degenerativa Mammalia Rodentia Sistema nervosio central patología Sistema nervioso patología Ungulata Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B17 Neurology / 002B17G Degenerative and inherited degenerative diseases of the nervous system. Leukodystrophies. Prion diseases
- Discipline
- Neurology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 1090200
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1097/00004647-200106000-00008 
https://dx.doi.org/10.1097/00004647-200106000-00008
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