Activation of Akt kinase inhibits apoptosis and changes in Bcl-2 and Bax expression induced by nitric oxide in primary hippocampal neurons
- Author
- MATSUZAKI, H1 2 ; TAMATANI, M1 2 ; MITSUDA, N1 ; NAMIKAWA, K3 ; KIYAMA, H2 3 ; MIYAKE, S.-I1 2 ; TOHYAMA, M1 2
[1] Department of Anatomy and Neuroscience, Osaka University Medical School, Osaka, Japan
[2] CREST, Japan Science and Technology, Hokkaido, Japan
[3] Department of Anatomy, Asahikawa Medical College, Hokkaido, Japan - Source
Journal of neurochemistry. 1999, Vol 73, Num 5, pp 2037-2046 ; ref : 1 p.1/4
- CODEN
- JONRA9
- ISSN
- 0022-3042
- Scientific domain
- Cell biology, histology; Neurology; Physiology, morphology
- Publisher
- Blackwell, Oxford
- Publication country
- United Kingdom
- Document type
- Article
- Language
- English
- Keyword (fr)
- Apoptose Azote monoxyde Facteur croissance IGF1 Hippocampe In vitro Protein kinase Rat Gène Bax Gène bcl2 Encéphale Enzyme Mammalia Rodentia Système nerveux central Transferases Vertebrata
- Keyword (en)
- Apoptosis Nitric oxide Insulin like growth factor 1 Hippocampus In vitro Protein kinase Rat Brain (vertebrata) Enzyme Mammalia Rodentia Central nervous system Transferases Vertebrata
- Keyword (es)
- Apoptosis Nitrógeno monóxido Factor crecimiento IGF1 Hipocampo In vitro Protein kinase Rata Encéfalo Enzima Mammalia Rodentia Sistema nervioso central Transferases Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H05 Ageing, cell death
- Discipline
- Molecular and cell biology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 1200584
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1046/j.1471-4159.1999.02037.x 
https://dx.doi.org/10.1046/j.1471-4159.1999.02037.x
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