Mechanism by which superoxide potentiates tubuloglomerular feedback
- Author
- YILIN REN1 ; CARRETERO, Oscar A1 ; GARVIN, Jeffrey L1
[1] Division of Hypertension and Vascular Research, Henry Ford Hospital, Detroit, Mich., United States - Source
Hypertension (Dallas, Tex. 1979). 2002, Vol 39, Num 2, pp 624-628 ; 2 ; ref : 35 ref
- CODEN
- HPRTDN
- ISSN
- 0194-911X
- Scientific domain
- Cardiology, blood circulation, phlebology
- Publisher
- Lippincott, Hagerstown, MD
/
Lippincott, Philadelphia, PA
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Animal Artériole Azote monoxyde Glomérule rénal Hypertension artérielle Nitric-oxide synthase Pathogénie Rat Appareil circulatoire pathologie Enzyme Mammalia Oxidoreductases Rodentia Vertebrata
- Keyword (en)
- Animal Arteriole Nitric oxide Renal glomerulus Hypertension Nitric-oxide synthase Pathogenesis Rat Cardiovascular disease Enzyme Mammalia Oxidoreductases Rodentia Vertebrata
- Keyword (es)
- Animal Arteriola Nitrógeno monóxido Glomérulo renal Hipertensión arterial Nitric-oxide synthase Patogenia Rata Aparato circulatorio patología Enzima Mammalia Oxidoreductases Rodentia Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B12 Cardiology. Vascular system / 002B12B Blood and lymphatic vessels / 002B12B05 Arterial hypertension. Arterial hypotension / 002B12B05A Experimental diseases
- Discipline
- Cardiology. Circulatory system
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 13509543
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1161/hy0202.103299 
https://dx.doi.org/10.1161/hy0202.103299
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