Constitutive c-Met signaling through a nonautocrine mechanism promotes metastasis in a transgenic transplantation model
- Author
- YANLIN YU1 ; MERLINO, Glenn1
[1] Molecular Genetics Section, Laboratory of Molecular Biology, National Cancer Institute, NIH, Bethesda, Maryland 20892-4264, United States - Source
Cancer research (Baltimore). 2002, Vol 62, Num 10, pp 2951-2956 ; ref : 59 ref
- CODEN
- CNREA8
- ISSN
- 0008-5472
- Scientific domain
- Medical oncology; Pharmacology drugs
- Publisher
- American Association for Cancer Research, Philadelphia, PA
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Animal transgénique Animal Antagoniste Expression génique Facteur croissance hépatocyte Gène onc cellulaire Invasion Migration Multiplication cellulaire Mécanisme action Mélanome malin Métastase Progression carcinogenèse Récepteur facteur croissance Souris Surexpression génique Transduction signal Gène NK2 Gène met Mammalia Rodentia Tumeur maligne Vertebrata
- Keyword (en)
- Transgenic animal Animal Antagonist Gene expression Hepatocyte growth factor C-Onc gene Invasion Migration Cell proliferation Mechanism of action Malignant melanoma Metastasis Tumor progression Growth factor receptor Mouse Gene overexpression Signal transduction Mammalia Rodentia Malignant tumor Vertebrata
- Keyword (es)
- Animal transgénico Animal Antagonista Expresión genética Factor crecimiento hepatocito Gen onc celular Invasión Migración Multiplicación celular Mecanismo acción Melanoma maligno Metástasis Progresión carcinogénesis Receptor factor crecimiento Ratón Sobreexpressión genética Transducción señal Mammalia Rodentia Tumor maligno Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes
- Discipline
- Molecular and cell biology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 13665953
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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