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Apoptotic signaling induced by immunomodulatory thalidomide analogs in human multiple myeloma cells: therapeutic implications

Author
MITSIADES, Nicholas; MITSIADES, Constantine S; POULAKI, Vassiliki; CHAUHAN, Dharminder; RICHARDSON, Paul G; HIDESHIMA, Teru; MUNSHI, Nikhil C; TREON, Steven P; ANDERSON, Kenneth C
Department of Adult Oncology, Dana-Farber Cancer Institute, the Department of Medicine, Harvard Medical School, and the Retina Research Laboratory, Massachusetts Eye and Ear Infirmary, Harvard Medical School, Boston, MA, United States
Source

Blood. 2002, Vol 99, Num 12, pp 4525-4530 ; ref : 35 ref

ISSN
0006-4971
Scientific domain
Medical oncology; Hematology
Publisher
The Americain Society of Hematology, Washington, DC
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Analogue Apoptose Cellule tumorale Homme Immunomodulation Myélome Mécanisme action Thalidomide Transduction signal Protéine inhibition apoptose Hémopathie maligne Immunoglobulinopathie Immunopathologie Lymphoprolifératif syndrome
Keyword (en)
Analog Apoptosis Tumor cell Human Immunomodulation Myeloma Mechanism of action Thalidomide Signal transduction Apoptosis inhibitory protein Malignant hemopathy Immunoglobulinopathy Immunopathology Lymphoproliferative syndrome
Keyword (es)
Análogo Apoptosis Célula tumoral Hombre Inmunomodulación Mieloma Mecanismo acción Talidomida Transducción señal Hemopatía maligna Inmunoglobulinopatía Inmunopatología Linfoproliferativo síndrome
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B06 Immunopathology / 002B06D Immunodeficiencies. Immunoglobulinopathies / 002B06D02 Immunoglobulinopathies

Discipline
Immunopathology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
13717480

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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