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The molecular mechanism in activation-induced cell death of an Ag-reactive B cell clone

Author
HAMANO, T1 ; IWASAKI, T1 ; OGATA, A1 ; HASHIMOTO, N1 ; KAKISHITA, E1
[1] Division of Clinical Immunology, Department of Internal Medicine, Hyogo College of Medicine, Nishinomiya, Hyogo 663-8501, Japan
Source

Clinical and experimental immunology (Print). 2002, Vol 128, Num 3, pp 436-443 ; ref : 46 ref

CODEN
CEXIAL
ISSN
0009-9104
Scientific domain
Biochemistry, molecular biology, biophysics; Immunology, immunopathology, allergology; General medicine general surgery
Publisher
Blackwell, Oxford
Publication country
United Kingdom
Document type
Article
Language
English
Keyword (fr)
Activation Animal Antigène Apoptose Forme soluble Hybridome Hyperoxyde Lignée cellulaire établie Lymphocyte B Mitochondrie Mort cellulaire Souris Stress oxydatif Gène bcl2 Lignée TPA-1 Gène onc cellulaire Mammalia Protooncogène Rodentia Vertebrata
Keyword (en)
Activation Animal Antigen Apoptosis Soluble form Hybridoma Hyperoxides Established cell line B-Lymphocyte Mitochondria Cell death Mouse Oxidative stress C-Onc gene Mammalia Protooncogene Rodentia Vertebrata
Keyword (es)
Activación Animal Antígeno Apoptosis Forma soluble Hibridoma Hiperóxido Línea celular establecida Linfocito B Mitocondria Muerte celular Ratón Estrés oxidativo Gen onc celular Mammalia Protooncogen Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A06 Fundamental immunology / 002A06C Immunobiology / 002A06C03 Lymphoid cells: ontogeny, maturation, markers, receptors, circulation and recirculation

Discipline
Fundamental immunology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
13729707

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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