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A decision between life and death during TNF-α-induced signaling

Author
GUPTA, Sudhir1
[1] Laboratories of Cellular and Molecular Immunology and Molecular Biology, Division of Basic and Clinical Immunology, University of California, Irvine, California 92697, United States
Source

Journal of clinical immunology. 2002, Vol 22, Num 4, pp 185-194 ; ref : 111 ref

CODEN
JCIMDO
ISSN
0271-9142
Scientific domain
Immunology, immunopathology, allergology
Publisher
Kluwer/Plenum, New York, NY
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Apoptose Caspase Facteur nécrose tumorale α Facteur transcription NFκB Homme Immunité Immunomodulation Lymphocyte T Mitochondrie Protein-tyrosine kinase Récepteur biologique Transduction signal Vieillissement Cytokine Enzyme Hydrolases Peptidases Transferases
Keyword (en)
Apoptosis Caspase Tumor necrosis factor α Transcription factor NFκB Human Immunity Immunomodulation T-Lymphocyte Mitochondria Protein-tyrosine kinase Biological receptor Signal transduction Ageing Cytokine Enzyme Hydrolases Peptidases Transferases
Keyword (es)
Apoptosis Caspase Factor necrosis tumoral α Factor transcripción NFκB Hombre Inmunidad Inmunomodulación Linfocito T Mitocondria Protein-tyrosine kinase Receptor biológico Transducción señal Envejecimiento Citoquina Enzima Hydrolases Peptidases Transferases
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A06 Fundamental immunology / 002A06C Immunobiology / 002A06C08 Modulation of the immune response (stimulation, suppression)

Discipline
Fundamental immunology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
13814458

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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