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Cardiac hypertrophy is not a required compensatory response to short-term pressure overload

Author
HILL, J. A1 2 3 ; KARIMI, M4 ; KUTSCHKE, W1 ; DAVISSON, R. L5 ; ZIMMERMAN, K3 ; ZHENGYI WANG1 ; KERBER, R. E1 ; WEISS, R. M1 3
[1] Division of Cardiovascular Diseases, Department of Internal Medicine, Department of Pharmacology, Department of Veterans Affairs, Department of Surgery, and Department of Anatomy and Cell Biology, University of Iowa College of Medicine, Iowa City, United States
[2] Department of Pharmacology, University of Iowa College of Medicine, Iowa City, United States
[3] Department of Veterans Affairs, University of Iowa College of Medicine, Iowa City, United States
[4] Department of Surgery, University of Iowa College of Medicine, Iowa City, United States
[5] Department of Anatomy and Cell Biology, University of Iowa College of Medicine, Iowa City, United States
Source

Circulation (New York, N.Y.). 2000, Vol 101, Num 24, pp 2863-2869 ; ref : 37 ref

CODEN
CIRCAZ
ISSN
0009-7322
Scientific domain
Cardiology, blood circulation, phlebology
Publisher
Lippincott Williams & Wilkins, Hagerstown, MD
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Animal Ciclosporine Echocardiographie Epreuve pharmacologique Exploration Fonction myocardique Fonction ventriculaire gauche Hypertrophie Inhibiteur Pathogénie Physiopathologie Pression sanguine Souris Surcharge Transduction signal Ventricule gauche Volume Calcineurine Appareil circulatoire pathologie Cardiopathie Exploration ultrason Mammalia Rodentia Vertebrata
Keyword (en)
Animal Ciclosporin Echocardiography Pharmacologic test Exploration Cardiac performance Left ventricle performance Hypertrophy Inhibitor Pathogenesis Pathophysiology Blood pressure Mouse Overload Signal transduction Left ventricle Volume Cardiovascular disease Heart disease Sonography Mammalia Rodentia Vertebrata
Keyword (es)
Animal Ciclosporina Ecocardiografía Prueba farmacológica Exploración Función miocárdica Función ventricular izquierda Hipertrofia Inhibidor Patogenia Fisiopatología Presión sanguínea Ratón Sobrecarga Transducción señal Ventrículo izquierdo Volumen Aparato circulatorio patología Cardiopatía Exploración ultrasonido Mammalia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B12 Cardiology. Vascular system / 002B12A Heart / 002B12A05 Myocarditis. Cardiomyopathies

Discipline
Cardiology. Circulatory system
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
1402424

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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