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BCR signals target p27Kip1 and cyclin D2 via the PI3-K signalling pathway to mediate cell cycle arrest and apoptosis of WEHI 231 B cells

Author
BANERJI, Lolita1 2 ; GLASSFORD, Janet1 2 ; LEA, Nicholas C3 ; THOMAS, N. Shaun B3 ; KLAUS, Gerry G. B4 ; LAM, Eric W-F1 2
[1] Ludwig Institute for Cancer Research and Section of Virology and Cell Biology, Imperial College School of Medicine at St Mary's, Norfolk Place, London W2 1PG, United Kingdom
[2] CRC Laboratories and Section of Cancer Cell Biology, Imperial College School of Medicine at Hammersmith Hospital, Du Cane Road, London W12 0NN, United Kingdom
[3] Department of Haematological Medicine, Guy's, King's, St. Thomas' School of Medicine, Rayne Institute, Leukaemia Sciences, Coldharbour Lane, London, United Kingdom
[4] Division of Cellular Immunology, National Institute for Medical Research Mill Hill, London, United Kingdom
Source

Oncogene (Basingstoke). 2001, Vol 20, Num 50, pp 7352-7367 ; ref : 1 p.1/4

ISSN
0950-9232
Scientific domain
Cell biology, histology; Medical oncology; Genetics
Publisher
Nature Publishing, Basingstoke
Publication country
United Kingdom
Document type
Article
Language
English
Keyword (fr)
Apoptose Arrêt Cible Cycle cellulaire Cycline D2 Lymphocyte B Signal Signalisation
Keyword (en)
Apoptosis Shutdown Target Cell cycle Cyclin D2 B-Lymphocyte Signal Signalling
Keyword (es)
Apoptosis Parada Blanco Ciclo celular Ciclina D2 Linfocito B Señal Señalización
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H05 Ageing, cell death

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
14154255

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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