Pascal and Francis Bibliographic Databases

Help

Permanent link : http://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=15005593

Export

Selection :

Mdm2 ligase dead mutants did not act in a dominant negative manner to re-activate p53, but promoted tumor cell growth

Author
SWAROOP, Manju1 ; YI SUN1
[1] Cancer Molecular Sciences, Pfizer Global Research and Development, Ann Arbor Laboratories, 2800 Plymouth Road, Ann Arbor, MI 48105, United States
Source

Anticancer research. 2003, Vol 23, Num 4, pp 3167-3174, 8 p ; ref : 32 ref

ISSN
0250-7005
Scientific domain
Medical oncology
Publisher
International Institute of Anticancer Research, Attiki
Publication country
Greece
Document type
Article
Language
English
Author keyword
Apoptosis E3 ubiquitin ligase Mdm2 dominant negative p53 transcription
Keyword (fr)
Adénocarcinome Glande mammaire Gène TP53 Gène onc cellulaire Gène suppresseur tumeur Homme Ligases Lignée cellulaire établie Multiplication cellulaire Mutation Protooncogène Ubiquitine Gène mdm2 Lignée MCF 7 Enzyme Glande mammaire pathologie Génétique Tumeur maligne
Keyword (en)
Adenocarcinoma Mammary gland TP53 Gene C-Onc gene Tumor suppressor gene Human Ligases Established cell line Cell proliferation Mutation Protooncogene Ubiquitin Enzyme Mammary gland diseases Genetics Malignant tumor
Keyword (es)
Adenocarcinoma Glándula mamaria Gen TP53 Gen onc celular Gen supresor tumor Hombre Ligases Línea celular establecida Multiplicación celular Mutación Protooncogen Ubiquitina Enzima Glándula mamaria patología Genética Tumor maligno
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B04 Tumors / 002B04G Tumor cell / 002B04G01 General aspects (metabolism, cell proliferation, established cell line...)

Discipline
Tumours
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
15005593

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

Searching the Web