What is the role of β-adrenergic signaling in heart failure?
- Author
- LOHSE, Martin J1 ; ENGELHARDT, Stefan1 ; ESCHENHAGEN, Thomas2
[1] Institute of Pharmacology, Wuerzburg, Germany
[2] Institute of Experimental and Clinical Pharmacology, Universitaetsklinikum Hamburg-Eppendorf, Germany - Source
Circulation research. 2003, Vol 93, Num 10, pp 896-906, 11 p ; ref : 184 ref
- CODEN
- CIRUAL
- ISSN
- 0009-7330
- Scientific domain
- Cardiology, blood circulation, phlebology
- Publisher
- Lippincott, Hagerstown, MD
- Publication country
- United States
- Document type
- Article
- Language
- English
- Author keyword
- G proteins apoptosis cardiac hypertrophy transgenic mice β-adrenergic receptors
- Keyword (fr)
- Animal transgénique Apoptose Fonction ventriculaire gauche Hypertrophie Insuffisance cardiaque Modèle animal Protéine G Récepteur β-adrénergique Souris Transduction signal Appareil circulatoire pathologie Cardiopathie Mammalia Rodentia Vertebrata
- Keyword (en)
- Transgenic animal Apoptosis Left ventricle performance Hypertrophy Heart failure Animal model G protein β-Adrenergic receptor Mouse Signal transduction Cardiovascular disease Heart disease Mammalia Rodentia Vertebrata
- Keyword (es)
- Animal transgénico Apoptosis Función ventricular izquierda Hipertrofia Insuficiencia cardíaca Modelo animal Proteína G Receptor β-adrenérgico Ratón Transducción señal Aparato circulatorio patología Cardiopatía Mammalia Rodentia Vertebrata
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B12 Cardiology. Vascular system / 002B12A Heart / 002B12A01 Heart failure, cardiogenic pulmonary edema, cardiac enlargement
- Discipline
- Cardiology. Circulatory system
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 15288487
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1161/01.RES.0000102042.83024.CA 
https://dx.doi.org/10.1161/01.RES.0000102042.83024.CA
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