NRAS and BRAF mutations arise early during melanoma pathogenesis and are preserved throughout tumor progression
- Author
- OMHOLT, Katarina1 ; PLATZ, Anton1 ; KANTER, Lena1 ; RINGBORG, Ulrik1 ; HANSSON, Johan1
[1] Cancer Centre Karolinska, Department of Oncology-Pathology, Radiumhemmet, Karolinska Hospital and Institute, Stockholm, Sweden - Source
Clinical cancer research. 2003, Vol 9, Num 17, pp 6483-6488, 6 p ; ref : 27 ref
- ISSN
- 1078-0432
- Scientific domain
- Medical oncology; Pharmacology drugs
- Publisher
- American Association for Cancer Research, Philadelphia, PA
- Publication country
- United States
- Document type
- Article
- Language
- English
- Keyword (fr)
- Carcinogenèse Gène onc Gène ras Homme Mutation Mélanome malin Métastatique Pathogénie Progression carcinogenèse Protein kinase Stade avancé Transduction signal Gène B-raf Gène N-ras Enzyme Transferases Tumeur maligne
- Keyword (en)
- Carcinogenesis Onc gene Ras gene Human Mutation Malignant melanoma Metastatic Pathogenesis Tumor progression Protein kinase Advanced stage Signal transduction Enzyme Transferases Malignant tumor
- Keyword (es)
- Carcinogénesis Gen onc Gen ras Hombre Mutación Melanoma maligno Metastásico Patogenia Progresión carcinogénesis Protein kinase Estadio avanzado Transducción señal Enzima Transferases Tumor maligno
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B08 Dermatology / 002B08A Tumors of the skin and soft tissue. Premalignant lesions
- Discipline
- Dermatology
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 15379943
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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