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NRAS and BRAF mutations arise early during melanoma pathogenesis and are preserved throughout tumor progression

Author
OMHOLT, Katarina1 ; PLATZ, Anton1 ; KANTER, Lena1 ; RINGBORG, Ulrik1 ; HANSSON, Johan1
[1] Cancer Centre Karolinska, Department of Oncology-Pathology, Radiumhemmet, Karolinska Hospital and Institute, Stockholm, Sweden
Source

Clinical cancer research. 2003, Vol 9, Num 17, pp 6483-6488, 6 p ; ref : 27 ref

ISSN
1078-0432
Scientific domain
Medical oncology; Pharmacology drugs
Publisher
American Association for Cancer Research, Philadelphia, PA
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Carcinogenèse Gène onc Gène ras Homme Mutation Mélanome malin Métastatique Pathogénie Progression carcinogenèse Protein kinase Stade avancé Transduction signal Gène B-raf Gène N-ras Enzyme Transferases Tumeur maligne
Keyword (en)
Carcinogenesis Onc gene Ras gene Human Mutation Malignant melanoma Metastatic Pathogenesis Tumor progression Protein kinase Advanced stage Signal transduction Enzyme Transferases Malignant tumor
Keyword (es)
Carcinogénesis Gen onc Gen ras Hombre Mutación Melanoma maligno Metastásico Patogenia Progresión carcinogénesis Protein kinase Estadio avanzado Transducción señal Enzima Transferases Tumor maligno
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B08 Dermatology / 002B08A Tumors of the skin and soft tissue. Premalignant lesions

Discipline
Dermatology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
15379943

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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