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Stimulation of lipolysis by tumor necrosis factor-α in 3T3-L1 adipocytes is glucose dependent: Implications for long-term regulation of lipolysis

Author
GREEN, Allan1 ; RUMBERGER, John M1 ; STUART, Charles A2 ; RUHOFF, Mary S1
[1] Bassett Research Institute, Bassett Healthcare, Cooperstown, New York, United States
[2] Department of Internal Medicine, James H. Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee, United States
Source

Diabetes (New York, NY). 2004, Vol 53, Num 1, pp 74-81, 8 p ; ref : 44 ref

CODEN
DIAEAZ
ISSN
0012-1797
Scientific domain
Endocrinology; Nutrition, obesity, metabolic disorders
Publisher
American Diabetes Association, Alexandria, VA
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Adipocyte Diabète Endocrinopathie Facteur nécrose tumorale α Glucose Lipolyse Long terme Cytokine
Keyword (en)
Adipocyte Diabetes mellitus Endocrinopathy Tumor necrosis factor α Glucose Lipolysis Long term Cytokine
Keyword (es)
Adipocito Diabetes Endocrinopatía Factor necrosis tumoral α Glucosa Lipólisis Largo plazo Citoquina
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A19 Vertebrates: skin, associated glands, phaneres, light organs, various exocrine glands (salt gland, uropygial gland...), adipose tissue, connective tissue

Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B21 Endocrinopathies / 002B21E Endocrine pancreas. Apud cells (diseases) / 002B21E01 Diabetes. Impaired glucose tolerance

Discipline
Endocrinopathies Vertebrates : skin, associated glands, phaneres, light organs, various exocrine glands, adipose tissue, connective tissue
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
15468006

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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