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Disruption of STAT3 signaling leads to tumor cell invasion through alterations of homotypic cell-cell adhesion complexes

Author
RIVAT, Christine1 ; DE WEVER, Olivier2 ; BRUYNEEL, Erik2 ; MAREEL, Marc2 ; GESPACH, Christian1 ; ATTOUB, Samir1
[1] INSERM U 482, Signal Transduction and Cellular Functions in Diabetes and Digestive Cancers, Hôpital Saint-Antoine, 184 Rue du Faubourg Saint-Antoine, 75571 Paris, France
[2] Laboratory of Experimental Cancerology, University Hospital, De Pintelaan 185, 9000 Gent, Belgium
Source

Oncogene (Basingstoke). 2004, Vol 23, Num 19, pp 3317-3327, 11 p ; ref : 1 p.1/4

ISSN
0950-9232
Scientific domain
Cell biology, histology; Medical oncology; Genetics
Publisher
Nature Publishing, Basingstoke
Publication country
United Kingdom
Document type
Article
Language
English
Author keyword
E-cadherin tumor suppressor gene EGF-R PI3-kinase Rho GTPases Wnt pathway β-catenin
Keyword (fr)
Adhérence Cadhérine E Carcinogenèse Caténine Cellule tumorale Gène suppresseur tumeur Kinase Mutation complète dGTPase Enzyme Esterases Hydrolases Transferases Triphosphoric monoester hydrolases
Keyword (en)
Adhesion E Cadherin Carcinogenesis Catenin Tumor cell Tumor suppressor gene Kinase Null mutation dGTPase Enzyme Esterases Hydrolases Transferases Triphosphoric monoester hydrolases
Keyword (es)
Adherencia Cadherina E Carcinogénesis Catenina Célula tumoral Gen supresor tumor Kinase Mutación nula dGTPase Enzima Esterases Hydrolases Transferases Triphosphoric monoester hydrolases
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
15724178

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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