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Angiotensin II impairs the insulin signaling pathway promoting production of nitric oxide by inducing phosphorylation of insulin receptor substrate-1 on Ser312 and Ser616 in human umbilical vein endothelial cells

Author
ANDREOZZI, Francesco1 ; LARATTA, Emanuela1 ; SCIACQUA, Angela1 ; PERTICONE, Francesco1 ; SESTI, Giorgio1
[1] Department of Clinical and Experimental Medicine, University Magna Græcia of Catanzaro, Italy
Source

Circulation research. 2004, Vol 94, Num 9, pp 1211-1218, 8 p ; ref : 29 ref

CODEN
CIRUAL
ISSN
0009-7330
Scientific domain
Cardiology, blood circulation, phlebology
Publisher
Lippincott, Hagerstown, MD
Publication country
United States
Document type
Article
Language
English
Author keyword
angiotensin II endothelium insulin nitric oxide
Keyword (fr)
Angiotensine II Appareil circulatoire Azote monoxyde Cellule endothéliale Endothélium Homme Insuline Mammalia Phosphorylation Récepteur biologique Transduction signal Veine ombilicale Hormone pancréatique Hormone peptide Octapeptide Système renin angiotensine Vertebrata
Keyword (en)
Angiotensin II Circulatory system Nitric oxide Endothelial cell Endothelium Human Insulin Mammalia Phosphorylation Biological receptor Signal transduction Umbilical vein Pancreatic hormone Peptide hormone Octapeptide Renin angiotensin system Vertebrata
Keyword (es)
Angiotensina II Aparato circulatorio Nitrógeno monóxido Célula endotelial Endotelio Hombre Insulina Mammalia Fosforilación Receptor biológico Transducción señal Vena umbilical Hormona pancreática Hormona péptido Octapéptido Sistema renin angiotensina Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A22 Vertebrates: cardiovascular system

Discipline
Vertebrates : cardiovascular system
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
16016686

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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