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The multifaceted role of mTOR in cellular stress responses

Author
PROUD, Christopher G1
[1] Division of Molecular Physiology, Faculty of Life Sciences, University of Dundee, Dow Street, Dundee, DD1 5EH, United Kingdom
Issue title
Bridge over broken ends: The cellular response to DNA breaks in health and disease
Author (monograph)
SHILOH, Yosef (Editor)1
[1] Department of Human Genetics and Molecular Medicine, Tel Aviv University, Sackler School of Medicine, Ramat Aviv, Tal Aviv 69978, Israel
Source

DNA repair. 2004, Vol 3, Num 8-9, pp 927-934, 8 p ; ref : 65 ref

ISSN
1568-7864
Scientific domain
Biochemistry, molecular biology, biophysics; Genetics
Publisher
Elsevier, Amsterdam
Publication country
Netherlands
Document type
Article
Language
English
Author keyword
Amino acids Apoptosis DNA damage Hypoxia Signaling mTOR
Keyword (fr)
Apoptose DNA Hypoxie Lésion Réparation Stress Facteur milieu Oxygène
Keyword (en)
Apoptosis DNA Hypoxia Lesion Repair Stress Environmental factor Oxygen
Keyword (es)
Apoptosis DNA Hipoxia Lesión Reparación Estrés Factor medio Oxígeno
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04C Molecular genetics / 002A04C07 Mutagenesis. Repair

Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H05 Ageing, cell death

Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A05 Microbiology / 002A05B Bacteriology / 002A05B05 Growth, nutrition, cell differenciation

Discipline
Microbiology Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
16042075

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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