Suppressed NF-κB and sustained JNK activation contribute to the sensitization effect of parthenolide to TNF-α-induced apoptosis in human cancer cells
- Author
- SIYUAN ZHANG1 ; LIN, Zhong-Ning2 ; YANG, Cheng-Feng1 ; XIANGLIN SHI3 ; ONG, Choon-Nam1 ; SHEN, Han-Ming1
[1] Department of Community, Occupational and Family Medicine, Faculty of Medicine (MD3), National University of Singapore, 16 Medical Drive, Singapore 117597, Singapore
[2] School of Public Health, Sun Yat-sen University, Guangzhou 510089, China
[3] Health Effects Laboratory Division, National Institute for Occupational Safety and Health, Morgantown, WV 26505, United States - Source
Carcinogenesis (New York. Print). 2004, Vol 25, Num 11, pp 2191-2199, 9 p ; ref : 46 ref
- CODEN
- CRNGDP
- ISSN
- 0143-3334
- Scientific domain
- Medical oncology; Genetics; Pharmacology drugs; Toxicology
- Publisher
- Oxford University Press, Oxford
- Publication country
- United Kingdom
- Document type
- Article
- Language
- English
- Keyword (fr)
- Activation Apoptose Cellule tumorale Facteur nécrose tumorale α Facteur transcription NFκB Homme Mort cellulaire Sensibilisation Tumeur maligne c-Jun N-terminal kinase
- Keyword (en)
- Activation Apoptosis Tumor cell Tumor necrosis factor α Transcription factor NFκB Human Cell death Sensitization Malignant tumor
- Keyword (es)
- Activación Apoptosis Célula tumoral Factor necrosis tumoral α Factor transcripción NFκB Hombre Muerte celular Sensibilización Tumor maligno
- Classification
- Pascal
- 002 Biological and medical sciences / 002B Medical sciences / 002B04 Tumors / 002B04E Carcinogenesis, carcinogens and anticarcinogens
- Discipline
- Tumours
- Origin
- Inist-CNRS
- Database
- PASCAL
- INIST identifier
- 16281798
Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS
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DOI
10.1093/carcin/bgh234 
https://dx.doi.org/10.1093/carcin/bgh234
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