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Programmed cell death protein 4 suppresses CDK1/cdc2 via induction of p21Waf1/Cip1

Author
GÖKE, R1 ; BARTH, P2 ; SCHMIDT, A2 ; SAMANS, B3 ; LANKAT-BUTTGEREIT, B1
[1] Clinical Research Unit for Gastrointestinal Endocrinology, 35033 Marburg, Germany
[2] Institute of Pathology, University of Marburg, 35033 Marburg, Germany
[3] Institute of Medical Biometry and Epidemiology, University of Marburg, 35033 Marburg, Germany
Source

American journal of physiology. Cell physiology. 2004, Vol 56, Num 6, pp C1541-C1546 ; ref : 33 ref

CODEN
AJPCDD
ISSN
0363-6143
Scientific domain
Cell biology, histology; Physiology, morphology
Publisher
American Physiological Society, Bethesda, MD
Publication country
United States
Document type
Article
Language
English
Author keyword
cell cycle tumor growth tumor suppressor gene
Keyword (fr)
Cellule tumorale Cycle cellulaire Côlon Facteur transcription Induction Mort cellulaire Multiplication cellulaire Poumon Prostate Protéine Tumeur Cyclin-dependent kinase Appareil digestif Appareil respiratoire Appareil urogénital Intestin
Keyword (en)
Tumor cell Cell cycle Colon Transcription factor Induction Cell death Cell proliferation Lung Prostate Protein Tumor Cyclin-dependent kinase Digestive system Respiratory system Urogenital system Gut
Keyword (es)
Célula tumoral Ciclo celular Colón Factor transcripción Inducción Muerte celular Multiplicación celular Pulmón Prostata Proteína Tumor Aparato digestivo Aparato respiratorio Aparato urogenital Intestino
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H04 Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
16288926

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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