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Dexamethasone enhances serum deprivation-induced necrotic death of rat C6 glioma cells through activation of glucocorticoid receptors

Author
MORITA, K1 ; ISHIMURA, K2 ; TSURUO, Y2 ; WONG, D. L3
[1] Department of Pharmacology, Tokushima University School of Medicine, 3-18-15 Kuramoto, Tokushima 770-8503, Japan
[2] Department of Anatomy and Cell Biology, Tokushima University School of Medicine, 3-18-15 Kuramoto, Tokushima 770-8503, Japan
[3] Nancy Pritzker Laboratory, Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, Stanford, CA 94305, United States
Source

Brain research. 1999, Vol 816, Num 2, pp 309-316 ; ref : 41 ref

CODEN
BRREAP
ISSN
0006-8993
Scientific domain
Neurology; Physiology, morphology
Publisher
Elsevier, Amsterdam / Elsevier, London / Elsevier, New York, NY
Publication country
Netherlands
Document type
Article
Language
English
Keyword (fr)
Cellule gliale Dexaméthasone Glucocorticoïde In vitro Lignée cellulaire Mort cellulaire Rat Récepteur hormonal Toxicité Lignée C6 Mammalia Névroglie Rodentia Vertebrata
Keyword (en)
Glial cell Dexamethasone Glucocorticoid In vitro Cell line Cell death Rat Hormonal receptor Toxicity Mammalia Neuroglia Rodentia Vertebrata
Keyword (es)
Célula glial Dexametasona Glucocorticoide In vitro Línea celular Muerte celular Rata Receptor hormonal Toxicidad Mammalia Neuroglia Rodentia Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A04 Molecular and cellular biology / 002A04H Cell physiology / 002A04H05 Ageing, cell death

Discipline
Molecular and cell biology
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
1648059

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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