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RASSF1A suppresses the c-jun-NH2-kinase pathway and inhibits cell cycle progression

Author
YOUNG MI WHANG1 ; YEUL HONG KIM1 2 3 ; JUN SUK KIM3 ; YOUNG DO YOO2 4
[1] Department of Internal Medicine and Brain Korea 21 Project for Biomedical Science, Korea, Republic of
[2] Genomic Research Center for Lung and Breast/Ovarian Cancers, Korea University Cancer Institute, Korea, Republic of
[3] 'Department of Internal Medicine, Korea University College of Medicine, Seoul, Korea, Republic of
[4] Graduate School of Medicine, Korea University College of Medicine, Seoul, Korea, Republic of
Source

Cancer research (Baltimore). 2005, Vol 65, Num 9, pp 3682-3690, 9 p ; ref : 29 ref

CODEN
CNREA8
ISSN
0008-5472
Scientific domain
Medical oncology; Pharmacology drugs
Publisher
American Association for Cancer Research, Philadelphia, PA
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Cycle cellulaire Gène onc cellulaire Inhibiteur Kinase Mitogen-activated protein kinase Transduction signal Enzyme Transferases
Keyword (en)
Cell cycle C-Onc gene Inhibitor Kinase Mitogen-activated protein kinase Signal transduction Enzyme Transferases
Keyword (es)
Ciclo celular Gen onc celular Inhibidor Kinase Mitogen-activated protein kinase Transducción señal Enzima Transferases
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B02 Pharmacology. Drug treatments / 002B02R Antineoplastic agents

Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B04 Tumors

Discipline
Pharmacological treatments Tumours
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
16724987

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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