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Synthetic protease inhibitor camostat prevents and reverses dyslipidemia, insulin secretory defects, and histological abnormalities of the pancreas in genetically obese and diabetic rats

Author
DONGMEI JIA1 ; TAGUCHI, Masashi1 ; OTSUKI, Makoto1
[1] Third Department of Internal Medicine, University of Occupational and Environmental Health, Japan, School of Medicine, Kitakyushu 807-8555, Japan
Source

Metabolism, clinical and experimental. 2005, Vol 54, Num 5, pp 619-627, 9 p ; ref : 37 ref

ISSN
0026-0495
Scientific domain
Endocrinology; Nutrition, obesity, metabolic disorders
Publisher
Elsevier, New York, NY
Publication country
United States
Document type
Article
Language
English
Keyword (fr)
Activité Animal Camostat Diabète type 2 Dyslipémie Défaut Dégénérescence Etat nutritionnel Expression génique Fibrose Hyperglycémie Hyperinsulinémie Inflammation Inhibiteur protease Inhibiteur synthétique Insuline Lipide Long terme Modèle Obésité Peptidases Puits RNA messager Rat Récepteur cholécystokinine Réponse Alimentation Comportement alimentaire Cytokine Endocrinopathie Enzyme Hormone pancréatique Hydrolases Inhibiteur enzyme Mammalia Métabolisme pathologie Pancréas endocrine Rodentia Serine endopeptidases Trouble nutrition Trypsin Vertebrata
Keyword (en)
Activity Animal Camostat Type 2 diabetes Dyslipemia Defect Degeneration Nutritional status Gene expression Fibrosis Hyperglycemia Hyperinsulinemia Inflammation Protease inhibitor Synthetic inhibitor Insulin Lipids Long term Models Obesity Peptidases Well Messenger RNA Rat Cholecystokinin receptor Response Feeding Feeding behavior Cytokine Endocrinopathy Enzyme Pancreatic hormone Hydrolases Enzyme inhibitor Mammalia Metabolic diseases Endocrine pancreas Rodentia Serine endopeptidases Nutrition disorder Trypsin Vertebrata
Keyword (es)
Actividad Animal Camostat Diabetes de tipo 2 Dislipemia Defecto Degeneración Estado nutricional Expresión genética Fibrosis Hiperglicemia Hiperinsulinemia Inflamación Inhibidor proteasa Inhibidor sintético Insulina Lípido Largo plazo Modelo Obesidad Peptidases Pozo RNA mensajero Rata Receptor colecistoquinina Respuesta Alimentación Conducta alimenticia Citoquina Endocrinopatía Enzima Hormona pancreática Hydrolases Inhibidor enzima Mammalia Metabolismo patología Páncreas endocrino Rodentia Serine endopeptidases Trastorno nutricíon Trypsin Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B22 Metabolic diseases / 002B22A Disorders of blood lipids. Hyperlipoproteinemia

Pascal
002 Biological and medical sciences / 002B Medical sciences / 002B22 Metabolic diseases / 002B22B Obesity

Discipline
Metabolic diseases
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
16789136

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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