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TNF, IFN-γ, and endotoxin increase expression of DMT1 in bronchial epithelial cells

Author
XINCHAO WANG1 ; GARRICK, Michael D2 ; FUNMEI YANG3 ; DAILEY, Lisa A4 ; PIANTADOSI, Claude A5 ; GHIO, Andrew J4
[1] Center for Environmental Medicine and Lung Biology, University of North Carolina, Chapel Hill, North Carolina, United States
[2] Department of Biochemistry, State University of New York at Buffalo, Buffalo, New York, United States
[3] Department of Cellular and Structural Biology, University of Texas Health Science Center, San Antonio, Texas, United States
[4] National Health and Environmental Effects Research Laboratory, Environmental Protection Agency, Research Triangle Park, North Carolina, United States
[5] Department of Internal Medicine, Duke University Medical Center, Durham, North Carolina, United States
Source

American journal of physiology. Lung cellular and molecular physiology. 2005, Vol 33, Num 1, pp L24-L33 ; ref : 52 ref

CODEN
APLPE7
ISSN
1040-0605
Scientific domain
Physiology, morphology; Pneumology
Publisher
American Physiological Society, Bethesda, MD
Publication country
United States
Document type
Article
Language
English
Author keyword
divalent metal transporter-1 interferon lipopolysaccharide membrane transporters tumor necrosis factor
Keyword (fr)
Appareil respiratoire Cellule épithéliale Endotoxine Facteur nécrose tumorale Interféron Lipopolyoside Mammalia Protéine transport Transporteur DMT1 Cytokine Toxine Vertebrata
Keyword (en)
Respiratory system Epithelial cell Endotoxin Tumor necrosis factor Interferon Lipopolysaccharide Mammalia Carrier protein DMT1 transporter Cytokine Toxin Vertebrata
Keyword (es)
Aparato respiratorio Célula epitelial Endotoxina Factor necrosis tumoral Interferón Lipopoliósido Mammalia Proteína transportador Citoquina Toxina Vertebrata
Classification
Pascal
002 Biological and medical sciences / 002A Fundamental and applied biological sciences. Psychology / 002A20 Vertebrates: respiratory system

Discipline
Vertebrates : respiratory system
Origin
Inist-CNRS
Database
PASCAL
INIST identifier
16872254

Sauf mention contraire ci-dessus, le contenu de cette notice bibliographique peut être utilisé dans le cadre d’une licence CC BY 4.0 Inist-CNRS / Unless otherwise stated above, the content of this bibliographic record may be used under a CC BY 4.0 licence by Inist-CNRS / A menos que se haya señalado antes, el contenido de este registro bibliográfico puede ser utilizado al amparo de una licencia CC BY 4.0 Inist-CNRS

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